Corrigendum to “Interaction between fortilin and transforming growth factor-beta stimulated clone-22 (TSC-22) prevents apoptosis via the destabilization of TSC-22” [FEBS Lett. 582 (2008) 1210–1218]
Article Outline
An unfortunate mistake occurred in the preparation of Fig. 5B. The correct figure and legend are given below.

Fig. 5.
Fortilin inhibits TSC-22 mediated apoptosis in human ovarian carcinoma cells, SKOV-3. Cell viability assay (A), DAPI staining (B) and caspase-3 activity assay (C) were performed on SKOV-3 ovarian cancer cells transfected with mock (an expression vector only without insert), fortilin, sifortilin, TSC-22 or siTSC-22, or co-transfected with TSC-22 and fortilin cDNAs, co-transfected with TSC-22 and sifortilin, or cotransfected with TSC-22 and sifortilin, or triple-transfected with fortilin, sifortilin and TSC-22. (A) Cell viability assay was quantified via tryptophan blue staining. Data are expressed as the means
±
S.E.M. (B) Cells were stained with DAPI to visualize DNA fragmentation for the apoptosis assay. Arrows indicate the observed DNA fragmentations. Size bar, 20
μm. The results are representative of three separate experiments. (C) Caspase-3 activity was measured using a microplate reader in fluorescence mode with an excitation wavelength of 400
nm and an emission wavelength of 505
nm. Enzyme activity was calculated and indicated as fluorescence in accordance with the formula provided by the manufacturer. Data are expressed as the means
±
S.E.M.
PII: S0014-5793(09)00103-3
doi:10.1016/j.febslet.2009.02.005
© 2009 Federation of European Biochemical Societies
Refers to article:
- Interaction between fortilin and transforming growth factor-beta stimulated clone-22 (TSC-22) prevents apoptosis via the destabilization of TSC-22 , 04 March 2008
